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Evidence Suggests an Infectious Link to Dementia

Dr. Russell Blaylock, M.D., writes: Pathologically, there are three things that are always seen in brains affected by Alzheimer’s disease: amyloid plaque, hyperphosphorylated tau protein, and signs of inflammation. There is dramatic evidence that immunoexcitotoxicity can cause all three of these pathological effects in the brain. Until recently, it was assumed that beta amyloid, the substance that forms amyloid plaque, was the cause of the brain degeneration associated with Alzheimer’s. But new evidence suggests that beta amyloid actually plays a lesser role, and that immunoexcitotoxicity is the primary mechanism of the disease. Doctors have known for a long time that some elderly people can have extensive amyloid deposits in their brain, yet still have perfectly normal memory, orientation, and other cognitive functions. It’s the type of amyloid that makes a difference. Tau protein is associated with neuron pathway function, so it is vital to brain function. When this protein is over phosphorylated, it begins to form clumps called neurofibrillary tangles. These block neuron pathways. (Newsmax.com)

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